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1
How would a T-cytotoxic cell respond if it encountered a T-helper cell that was infected with a virus?
2
Why would it be unwise to suppress the symptom of a "swollen gland"?
3
Would a denatured antigen be expected to have the same epitopes as its native (undenatured) counterpart?
4
In opsonization with IgG, why would it be important that IgG react with the antigen before a phagocytic cell recognizes the antibody molecule?
5
If the heavy chain of an antibody consists of four domains, each approximately 110 amino acids long, how much DNA would be required to encode 109 separate heavy chain genes?
6
Why should B cells residing in the mucosal-associated lymphoid tissue produce IgA?
7
Why would a person who has AIDS be more susceptible to the bacterium that causes tuberculosis?
8
What selective advantage would a virus have if it interferes with the process of antigen presentation?
9
How could imprecise joining be considered a type of frameshift mutation?







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